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    Reproductive and Developmental Toxicity. Since February 2015 di(2‐ethylhexyl) phthalate (DEHP) di‐n‐butyl phthalate (DBP) and butylbenzyl phthalate (BBP) may only be used after prior authorization (EU 2011). Documentation was published for DEHP in 2002 documentation "Di(2‐ethylhexyl)phthalate (DEHP)" 2009 followed by a supplement in 2015 with new data for the mechanism of action

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  • Classification and Risk Assessment of Chemicals The Case

    In contrast DEHP administration throughout gestation in CD-I mice resulted in an increased incidence of malformations at doses which produced maternal and other embryofetal toxicity (0.10 and 0

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  • (PDF) Effects of Di(2-ethylhexyl) Phthalate on Regulation

    Effects of DEHP on Testes and Epididymis Weights Four-week old rats were treated with DEHP (250 500 or 750 mg/kg) for 30 days. The data is reported as the mean ± S.E. for 6 animals/group.

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  • Comparison of in vitro hormone activities of selected

    2009-12-1 · Co-administration of DBP and DEHP in utero significantly reduced testosterone levels in rat foetal testis (Martino-Andrade et al. 2008). When MBP was measured in urine with high-performance liquid chromatography and tandem mass spectrometry the results suggested that there were dose–response relationships between MBP and sperm motility and

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  • Genetic resistance to DEHP-induced transgenerational

    2019-6-10 · Di(2-ethylhexyl)phthalate (DEHP) interferes with hormones signaling pathways (SHP). C57BL/6J mice prenatally exposed to 300 mg/kg/day DEHP develop a testicular dysgenesis syndrome (TDS) at adulthood but similarly-exposed FVB/N mice are not affected. Here we aim to understand the reasons behind this drastic difference that should depend on the genome of the strain.

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    Administration of Potentially Antiandrogenic Pesticides (Procymidone Linuron Iprodione Chlozolinate p p -DDE and Ketoconazole) and Toxic Substances (Dibutyl- And Diethylhexyl Phthalate PCB 169 and Ethane Dimethane Sulphonate) During Sexual Differentiation Produces Diverse Profiles of Reproductive Malformations in the Male Rat

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  • Di-(2-propylheptyl) phthalate (DPHP) and its metabolites

    2016-9-30 · Di-(2-propylheptyl) phthalate (DPHP) does not act as a reproductive toxicant or endocrine disruptor in contrast to other phthalates. Considering adverse effects of phthalates to be linked to their metabolism it was the aim of the present study to investigate in the rat the blood burden of DPHP and its metabolites as a basis for understanding the toxicological behavior of DPHP.

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